Xanthic: Hereditary or Anomaly?

[elting44]

Is Xanthism expressed genetically from the passing of a dominant/recessive gene from parent to offspring, or is it a mutation?

Also, Is xanthism identifiable from birth or does it take time (until the fish reaches adulthood) to develop?

I have tried to research this subject, but I am coming up with limited and conflicting answers. Can someone give me a definitive answer?

 

[Jesse]

My guess is that, like albinism, it is hereditary in most species, passing from one generation to the next via recessive genes with only homozygous xanthic genotypes exhibiting the phenotypic xanthism.

 

[js302]

Is Xanthism expressed genetically from the passing of a dominant/recessive gene from parent to offspring, or is it a mutation?

could be a mutation but this is highty improbable but not impossible as the mutation would have to occur at a very particular gene locus/position. or at some other point knocking out another gene(s) thus bringing about xanthic effects.

Also, Is xanthism identifiable from birth or does it take time (until the fish reaches adulthood) to develop?

newly hatched fry are so small and dont really express detailed pigmentation until later. so some growing out would be needed. xanthism MAY be identifyable indirectly eg with ebjd's where i think ebjd fry actually grow slower and complete less well for food than their 'normal' counterparts.

 

[elting44]

I was wondering if they could be ID'ed as Xenomorphic while juvenile, 1"-2", or if the pattern takes until adulthood to present.

Also, I found Xanthism is an autosomal recessive mutation, so it is hereditary. The study said that the xanthic form is a type of tyrosinase-positive partial albinism in Snakes.

So if that is the case in fish as well, one can assume that if Xanthic Male and Non-xanthic female that carries the recessive gene were to produce offspring, roughly 50% would have Xanthism.

Is this correct?

 

[js302]

I was wondering if they could be ID'ed as Xenomorphic while juvenile, 1"-2", or if the pattern takes until adulthood to present.

Also, I found Xanthism is an autosomal recessive mutation, so it is hereditary. The study said that the xanthic form is a type of tyrosinase-positive partial albinism in Snakes.

So if that is the case in fish as well, one can assume that if Xanthic Male and Non-xanthic female that carries the recessive gene were to produce offspring, roughly 50% would have Xanthism.

Is this correct?

1 to 2 inches may be ok for IDing.

im not sure about 50 50. maybe a lot less.

in a simple recessive dominant mendelian kinda genetic framework, if the father is double recessive (xanthic) and the mother is double positive for normal pigmentation, all offspring would be positive for the recesssive gene but not express it, they'd be like carriers. two heterozygous individuals must now cross to produce 25% double recesssive offspring.

it may be a bit complicated tho, especially if the xanthic individual expresses xanthism due to an alele mutation at a gene locus.

 

[elting44]

I see, I got 50% from taking a double recessive (xanthic), against a Non-xanthic recessive carrier. Aa : aa, To result in 2 x Aa and 2 x aa or 50/50. Thank you though, does anyone know offhand if xanthism/positvie partial ablinism is caused by Alele mutation. I am wishing I hadn't sold my biology book back to the bookstore!