Also just remembered, how would a small "sheet" of fat be able to push the eye out? Wouldnt the fat have to be a solid mass and not a layer like thing? Also, why would only the top of the eye be pushed out from the socket? Wouldn't the whole eye be pushed out if fat was accumulated behind it? Some pictures here show a more severe case of drop eye.
These pictures were taken after the owner came back from a long weekend stating that the aro's were perfectly fine before he left. How would fat be able to accumulate in such short amounts of time? What kind of event, other than jumping into the lid, could've happened in these short days? This already takes out quite a few theories. Most theories require long periods of time, saying that DE will slowly develop becoming more and more severe, but how many of us have actually experienced something like that? Fat accumulation would take months to happen, thats for sure. Also, with theories such as, lack of excercise, it would also take quite a bit of time to actually drop, and would definitely not happen after 3-4 days. Also, if this aro was left alone for the long weekend with no one in the house, I doubt the owner would keep his lights on for 3-4 days, if this was the case, we don't even need to bother with the light reflection theories. Theres also some people saying that DE is caused by water params, fluctuations, and cold water water changes. If the owner was gone for the long weekend, how would he have even altered the water? The water would've been untouched at all, thus throwing out those theories as well. We also need to take into consideration, why would only one eye be affected instead of both at the same time? Also, looking at the first picture, we can clearly see the eye instead of drooping out, has actually fallen INTO the socket. If there was fat at the back of the eye pushing it out to droop, why is there space for this to happen? As you can see there is a sort of thin membrane holding the eye in both pictures, which i suspect is what the people cut out in the video posted as you obviously cannot get to what is behind the eye since this membrane like thing is blocking off the back. My question now, other than what forms DE, is, if this was really what they cut out, how the heck did that aro keep its eyes in the socket? Anyways, back on topic, the only theories now that I see possible are genetics and head trauma. Now heres where the tricky part comes in. There have been many people (Bderick67 and me included) that have seen DE develop within hours of head trauma. This, along with genetics would be able to explain everything above, plus why one eye is affect in most cases. Now the problem is, why do different species have different chances of getting DE? Going from highest chance to lowest change is:
1. Silver
2. Asian
3. Jar/Lei
4. Black
5. African
2 and 3 may be swapped, but from what i can see this is generally the order. The only trend I can see here is, it goes down the line as to how commonly found the aros are. Silver being very common, and african being very rare. So do silvers really have a genetic trait that makes them more prone to DE (supporting genetics) or is it just because silvers are most common, so we mostly hear and see of silvers having DE? This would suggest that all aros have the same amount of chance, and are capable of getting DE, but because we see and hear more of one species, thus we assume they are more suceptible to DE. Although possible, it is also quite improbable that genetics cause an aro's eye to drop over the course of a couple days, which would make head trauma seem like a more probable cause. Anyone else want to add on or rebut?