This is more than crude fat and crude protein percentages, mineral and trace mineral dietary numbers. You are given that this is fish tissue which gives you an excellent idea of the amino acid profile and its digestibility.
The amino acid profile of fish tissue varies from species to species, if you can show me that goldfish have a superior amino acid profile compared to a food that has the bulk of its protein comprised from Herring, Antarctic Krill (Euphausia superba), and Squid, I'm all ears. The NRC's (National Research Council's) amino acid stats for the 4 most popular forms of fish meal, which include Herring meal, Anchovy Meal, Whitefish meal, and Menhaden meal, clearly demonstrate that not all fish are equal in crude protein, or amino acid content. The same applies to fatty acid profiles, the fact that a fat is unprocessed, does not equate to it receiving a gold label in quality.
BTW - I not only read the stats on goldfish & minnows from that paper you cited previously, I linked to it in the gar nutrition discussion that took place last year that I mentioned in my original comment. None of this is news to me, Rich.

Crude percentages of protein and fat on a feed label aren't very meaningful unless you know what the contents are. Even then you don't know the quality of the product. Fishmeal comes in different grades and freshness. We are talking whole, live fish and that is a lot of information in that statement. As for fat content, its fresh unprocessed fish oil. These are the gold standards in fish diets. We are not talking beef tallow, meat and bone meal, feather meal or dried blood. Its whole fish.
Now tell me something that I don't know. Again, this is old news. No one here is comparing bulk generic farm feed that may contain ingredients such as beef tallow, meat & bone meal, feather meal or blood meal, with goldfish, or anything else. There are commercial diets available in today's market that utilize nothing but the best of the best raw ingredients, those foods are simply not going to be economical for a commercial producer such as yourself. At a hobbyist level their costs are not a huge factor for most people that want the best overall nutrient levels that they can give their fish. Not just protein, and not just fat. Again, the only compromise that I see being made is that by the commercial farmer, who due to their large scale commercial applications must seek lower cost alternative feeds.
Yes, fish in general have a low tolerance for high carbohydrate diets. Piscovers are especially prone to diseases associated with high carbohydrate diets. There was a study in the Journal of The World Aquaculture Society (Vol. 33, No.4) where largemouth bass suffered serious liver damage and high mortality rates as a consequence of being fed a trout diet with excessive carbohydrate levels (>27%). I dont know if you intended to make my case here for me or not.
I've been preaching against high carb inclusion rates in diets designed for fish for years, there is no argument there, but the inclusion rate of carbs can easily be controlled in a pellet feed. That my friend is entirely up to each manufacturer. The pellet food that I feed has a low carb content, and is in no way detrimental to the health of a piscivore.
I don't know anyone that keeps, or breeds piscivores such as Cyphotilapia frontosa, that feeds goldfish.
I disagree with you on this point. The bulk of feeders sold are merely fry. The balance of their free metabolic energy goes to tissue development. Not until fish exceed 100 grams will they begin to sexually mature and begin laying on fat stores. So while there will be some variation at the smaller sizes (aprox. 1 gram), I don't believe the numbers would range widely.
For a starter the proximate analysis that you are using for the foundation of your argument was based on the analysis of a single goldfish. No mention of species, size, weight, life stage, or anything else. Furthermore a fish does not have to be sexually mature to potentially have excessive lipid deposition. This would be highly dependent on exactly what & how much a goldfish is fed.
As an example;
http://www.tandfonline.com/doi/abs/10.1577/A03-035.1
A 12-week feeding trial was conducted to evaluate growth performance and hepatocyte changes in juvenile African cichlids Pseudotropheus socolofi and Haplochromis ahli fed commercial diets commonly used on cichlid farms in south Florida. Fish were fed either a trout starter pellet diet (TP diet; 52% crude protein, 17% lipid) or a mixed flake feed diet (fish flake [FF] diet) (47% crude protein, 7% lipid). For both species, growth was significantly greater (P < 0.05) in fish fed the TP diet than in fish fed the FF diet, H. ahli administered the FF diet exhibiting the slowest growth. The hepatosomatic index was not significantly different among treatments (P > 0.1). Histological examination revealed severe vacuolation of hepatocytes in P. socolofi fed the TP diet and moderate vacuolation in H. ahli fed the TP diet. Moderate vacuolation of hepatocytes was observed in P. socolofi fed the FF diet, and only mild vacuolation was found in H. ahli fed the FF diet. The lipid-rich TP diet may be suitable for commercial production of juvenile African cichlids up to 12 weeks of age, but prolonged feeding may result in excess lipid deposition and necrosis of the liver. Feeds like the FF diet, which produced slower growth but lower lipid deposition in livers, may be more suitable as a maintenance diet for cichlids in the home aquarium.
The fish used in this study were only 4 weeks old when the feed trial began, and averaged 0.06 g in weight, and 1.5 cm in total length.
The juvie H. ahli (S. fryeri) used in this study fared much better being a carnivore/piscivore, but it still showed a lipid accumulation in the hepatocytes. (<50%) The P. socolofi had extensive lipid accumulation when fed the TP diet.
This study came about after some cichlid farms in south FL suffered from some large mortalities in both 1998 & 1999. When the dead fish were examined they showed fatty infiltration of the liver, heavy vacuolation, and severe necrosis of the liver, pancreas, and spleen.
It was suggested to the farms that they replace ther feeds with one that had a lower lipid content (less than 10%) and supplement the feed with a vitamin premix. Clinical signs in the affected farms were resolved after implementation of these recommendations.
And I am somehow supposed to believe that a "goldfish" couldn't accumulate fat, when a juvenile carnivore/piscivore such as an S. fryeri did just that, in only 12 weeks.
Hmmmmmmm.
In my mind the entire thiaminase issue is a no brainer. When there are species of feeder fish that are known to not produce this B1 destroying enzyme, it seems rather obvious to me that one should go the safe route & feed those species, vs one such as goldfish that are known producers of thiaminase. As you said, at this point not much is even known about thiaminase, other than we do know that it has the potential to destroy B1.
Knowing that, why would one want to play Russian roulette with their fishes health, when safer alternatives are available? It makes absolutely no sense.
Unless of course one is simply looking for ways to gain support for a potential business venture of breeding/raising goldfish feeders.
