HITH is caused by stress, and in a glass box stress can come in many forms, for many different species of fish.
In the case of blackwater species I agree with both Matt, and Tom, in that those species found in acidic blackwater environments do not always cope well with various parasites/pathogens found in non blackwater environments.
Why do some fish get HITH while others kept in the same tank, and fed the exact same diet do not? I posted the following in a past discussion on fish health .....
IMO the reason for this is due to the fact that all fish are individuals, and while they can have individual behaviors & temperaments, I also believe that each fish can only tolerate individual levels of stress before they weaken enough to become susceptible to these stressors. You'll typically find that the same holds true for most organisms on our planet, including humans.
Just as one can have genetic runts, I also believe that one can end up with fish that have genetically weak immune systems, or at least weaker than normal. This would explain why one fish in a tank full of (any species), can end up with fungus, or pop eye, or HITH, or whatever, while other fish in the same tank have no health issues whatsoever. Or why some fish survive an ammonia spike, while others drop like flies. Or why only one female in a tank full of breeders ends up egg bound, while no other females ever exhibit this problem.
In the wild Mother Nature has a way of dealing with fish such as this, but what if some of these fish are collected, and exported? How many people cull WC fish? Most breeders will do almost anything to save a prized egg bound female, yet in the wild that fish would either pull through on its own, or else .....
Overuse of medication can also cause an immune system to become severely compromised. Many exporters & importers dose the fish with meds shortly after they are collected or received, again with tranqs when the fish are shipped, while many hobbyists dose the fish yet again with meds when they arrive to their new home. (as a precautionary step) Already the fish have the odds stacked against them, and all it takes is one single weak link to start a snowball effect in ones tank.
Perhaps in some cases, even when we do everything right, it ultimately all boils down to nothing more than a roll of the dice with certain individual fish.
..................................................
The actual pathogen that has been identified as being most commonly associated with HITH disease is Spironucleus vortens, a pathogen that I have discussed in great detail in a number of past threads, including a few that were later made stickies here on MFK. From one of those past discussions;
That info came from a 200+ page paper on S. vortens and Angelfish, which can be found in the following link.
http://scholar.lib.vt.edu/theses/available/etd-120399-140825/unrestricted/ANGEL.PDF
HTH
In the case of blackwater species I agree with both Matt, and Tom, in that those species found in acidic blackwater environments do not always cope well with various parasites/pathogens found in non blackwater environments.
Why do some fish get HITH while others kept in the same tank, and fed the exact same diet do not? I posted the following in a past discussion on fish health .....
IMO the reason for this is due to the fact that all fish are individuals, and while they can have individual behaviors & temperaments, I also believe that each fish can only tolerate individual levels of stress before they weaken enough to become susceptible to these stressors. You'll typically find that the same holds true for most organisms on our planet, including humans.
Just as one can have genetic runts, I also believe that one can end up with fish that have genetically weak immune systems, or at least weaker than normal. This would explain why one fish in a tank full of (any species), can end up with fungus, or pop eye, or HITH, or whatever, while other fish in the same tank have no health issues whatsoever. Or why some fish survive an ammonia spike, while others drop like flies. Or why only one female in a tank full of breeders ends up egg bound, while no other females ever exhibit this problem.
In the wild Mother Nature has a way of dealing with fish such as this, but what if some of these fish are collected, and exported? How many people cull WC fish? Most breeders will do almost anything to save a prized egg bound female, yet in the wild that fish would either pull through on its own, or else .....
Overuse of medication can also cause an immune system to become severely compromised. Many exporters & importers dose the fish with meds shortly after they are collected or received, again with tranqs when the fish are shipped, while many hobbyists dose the fish yet again with meds when they arrive to their new home. (as a precautionary step) Already the fish have the odds stacked against them, and all it takes is one single weak link to start a snowball effect in ones tank.
Perhaps in some cases, even when we do everything right, it ultimately all boils down to nothing more than a roll of the dice with certain individual fish.
..................................................
The actual pathogen that has been identified as being most commonly associated with HITH disease is Spironucleus vortens, a pathogen that I have discussed in great detail in a number of past threads, including a few that were later made stickies here on MFK. From one of those past discussions;
"In freshwater fish, Spironucleus sp. has been reported in cichlids, including angelfish, and cyprinids. Spironucleus elegans has been found causing disease in angelfish (Pterophyllum scalare) in Europe (Kulda and Lom, 1964b), while Spironucleus vortens has been also reported from the intestinal lumen of angelfish bred in Florida (Poynton et al., 1995). They can be found in the intestinal tracts of healthy fish and they may invade the body of the host and are capable of causing severe parasitemia under stress conditions (Molnár, 1974).
Hexamita are considered commensal organisms but can be pathogenic under various stressful conditions such as nutritional inadequacies, suboptimal water quality, crowding, poor sanitation and infections from other pathogens or parasites (Specht et al., 1989; Uzmann et al., 1965)
Although Hexamita and Spironucleus cause disease in several economically important fish species in many regions of the world, their pathogenicity is not well known (Woo and Poynton, 1995). It has been believed that the effects and the severity of diplomonad infection are dependent on fish size, tissue site infested, environmental conditions, stocking density, season and stress factors (Allison, 1963; Buchmann and Uldal, 1996; Mo et al., 1990; Uldal and Buchmann, 1996). Fish infected with Hexamita salmonis exhibited clinical signs correlated to their body length and weight (Uldal and Buchmann, 1996). This supports the study of Allison (1963) that large trout, more than 3 or 4 inches long, showed a smaller effect from Hexamita infection than smaller trout.
It is believed that the organisms invade the intestinal epithelium and disseminate to other tissues when the hosts resistance has been suppressed (Woo and Poynton, 1995). It has been suggested that only Spironucleus spp. cause systemic infection because they can invade intestinal mucosa and disseminate to other tissues (Siddall et al., 1992). Evidence of systemic spironucleosis was reported in cyprinids and aquarium fishes (Molnár, 1974), and in salmonids (Mo et al., 1990; Poppe et al., 1992; Sterud et al., 1997).
It has been suggested that only Spironucleus spp. cause systemic infection because they can invade intestinal mucosa and disseminate to other tissues (Siddall et al., 1992). Evidence of systemic spironucleosis was reported in cyprinids and aquarium fishes (Molnár, 1974), and in salmonids (Mo et al., 1990; Poppe et al., 1992; Sterud et al., 1997). In cyprinids, many Spironucleus sp. were found in the gut where they caused reddening of the mucous membrane, brownish-gray discoloration and necrosis of liver, and haemorrhagic enteritis. In aquarium fishes, the parasite has been reported to cause losses in angelfish stock of up to 50% of the population. The infected fishes showed reddening of the skin, and haemorrhages and ulcerations in the region of the head (Molnár, 1974). The parasites were found in the gut, gallbladder and visceral organs. Spironucleus sp. were also found in large numbers in the blood and the muscle of infected fish."
That info came from a 200+ page paper on S. vortens and Angelfish, which can be found in the following link.
http://scholar.lib.vt.edu/theses/available/etd-120399-140825/unrestricted/ANGEL.PDF
HTH